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Capecitabine plus bevacizumab

GERCOR - Multidisciplinary Oncology Cooperative Group · Phase 3 active Small molecule

Capecitabine is a prodrug that converts to fluorouracil to inhibit thymidylate synthase and disrupt DNA synthesis, while bevacizumab is a monoclonal antibody that blocks VEGF to inhibit tumor angiogenesis.

Capecitabine is a prodrug that converts to fluorouracil to inhibit thymidylate synthase and disrupt DNA synthesis, while bevacizumab is a monoclonal antibody that blocks VEGF to inhibit tumor angiogenesis. Used for Metastatic colorectal cancer, Metastatic breast cancer.

At a glance

Generic nameCapecitabine plus bevacizumab
Also known ascapecitabine, Avastin
SponsorGERCOR - Multidisciplinary Oncology Cooperative Group
Drug classAntimetabolite plus monoclonal antibody
TargetThymidylate synthase (capecitabine); VEGF (bevacizumab)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Capecitabine is metabolized to 5-fluorouracil, which inhibits thymidylate synthase and incorporates into DNA/RNA to impair cancer cell replication. Bevacizumab binds vascular endothelial growth factor (VEGF), preventing its interaction with VEGF receptors on endothelial cells and thereby blocking new blood vessel formation that tumors depend on for growth and metastasis. The combination provides complementary cytotoxic and anti-angiogenic effects.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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