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Capecitabine and Bevacizumab

South Eastern European Research Oncology Group · Phase 2 active Small molecule

Capecitabine is a prodrug that converts to fluorouracil to inhibit thymidylate synthase and disrupt DNA synthesis, while bevacizumab is a monoclonal antibody that blocks VEGF to inhibit tumor angiogenesis.

Capecitabine is a prodrug that converts to fluorouracil to inhibit thymidylate synthase and disrupt DNA synthesis, while bevacizumab is a monoclonal antibody that blocks VEGF to inhibit tumor angiogenesis. Used for Metastatic colorectal cancer, Metastatic breast cancer, Gastric cancer.

At a glance

Generic nameCapecitabine and Bevacizumab
Also known asBevacizumab (Avastin ), Capecitabine (Xeloda)
SponsorSouth Eastern European Research Oncology Group
Drug classFluoropyrimidine antimetabolite + VEGF inhibitor (monoclonal antibody)
TargetThymidylate synthase (capecitabine/5-FU) and VEGF (bevacizumab)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 2

Mechanism of action

Capecitabine is an oral fluoropyrimidine carbamate that is metabolized to 5-fluorouracil (5-FU), which inhibits thymidylate synthase and incorporates into RNA and DNA to impair cancer cell proliferation. Bevacizumab is a humanized monoclonal antibody targeting vascular endothelial growth factor (VEGF), preventing new blood vessel formation that tumors depend on for growth and metastasis. The combination leverages chemotherapy-induced cytotoxicity with anti-angiogenic effects.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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