Last reviewed · How we verify

Camrelizumab+Apatinib+Eribulin

Sun Yat-Sen Memorial Hospital of Sun Yat-Sen University · Phase 3 active Small molecule

This combination therapy enhances anti-tumor immunity by blocking PD-1 checkpoint inhibition while simultaneously inhibiting angiogenesis and disrupting microtubule dynamics to suppress tumor growth.

This combination therapy enhances anti-tumor immunity by blocking PD-1 checkpoint inhibition while simultaneously inhibiting angiogenesis and disrupting microtubule dynamics to suppress tumor growth. Used for Advanced or metastatic breast cancer (phase 3 investigation).

At a glance

Generic nameCamrelizumab+Apatinib+Eribulin
SponsorSun Yat-Sen Memorial Hospital of Sun Yat-Sen University
Drug classPD-1 inhibitor + tyrosine kinase inhibitor + microtubule inhibitor combination
TargetPD-1 (camrelizumab), VEGFR-2 (apatinib), microtubule dynamics (eribulin)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Camrelizumab is a PD-1 inhibitor that restores T-cell-mediated anti-tumor immunity by blocking the PD-1/PD-L1 axis. Apatinib is a tyrosine kinase inhibitor targeting VEGFR-2 that suppresses tumor angiogenesis and vascular endothelial growth. Eribulin is a microtubule dynamics inhibitor that disrupts mitotic spindle formation and induces apoptosis in cancer cells. The triple combination aims to synergistically attack tumors through immune activation, anti-angiogenesis, and direct cytotoxicity.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: