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Calcineurin inhibitor therapy

Andrew B Adams · Phase 3 active Small molecule

Calcineurin inhibitors suppress T-cell activation by blocking the phosphatase calcineurin, preventing the dephosphorylation and nuclear translocation of NFAT transcription factors required for cytokine production.

Calcineurin inhibitors suppress T-cell activation by blocking the phosphatase calcineurin, preventing the dephosphorylation and nuclear translocation of NFAT transcription factors required for cytokine production. Used for Organ transplant rejection prophylaxis, Autoimmune and inflammatory disorders.

At a glance

Generic nameCalcineurin inhibitor therapy
Also known astacrolimus, cyclosporine
SponsorAndrew B Adams
Drug classCalcineurin inhibitor
TargetCalcineurin (protein phosphatase 2B)
ModalitySmall molecule
Therapeutic areaImmunology
PhasePhase 3

Mechanism of action

These agents inhibit calcineurin, a serine/threonine phosphatase essential for T-cell receptor signaling. By blocking calcineurin, they prevent the activation and proliferation of T lymphocytes, thereby reducing immune-mediated inflammation and rejection responses. This mechanism makes them effective immunosuppressants in transplantation and autoimmune conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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