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Bupivacaine-epinephrine

Lumbini Medical College · FDA-approved active Small molecule

Bupivacaine blocks sodium channels in nerve cell membranes to produce local anesthesia, while epinephrine causes vasoconstriction to prolong anesthetic duration and reduce systemic toxicity.

Bupivacaine blocks sodium channels in nerve cell membranes to produce local anesthesia, while epinephrine causes vasoconstriction to prolong anesthetic duration and reduce systemic toxicity. Used for Local and regional anesthesia for surgical procedures, Infiltration anesthesia, Nerve blocks.

At a glance

Generic nameBupivacaine-epinephrine
Also known asBupivacaine 0.5% with adrenaline vasoconstrictor 1: 200,000, Marcain-adrenalin (combination drug), Marketing Authorisation Holder: AstraZeneca, Marketing Authorisation number: 10213, Exparel
SponsorLumbini Medical College
Drug classLocal anesthetic with vasopressor
TargetVoltage-gated sodium channels; alpha-1 and beta-2 adrenergic receptors
ModalitySmall molecule
Therapeutic areaAnesthesia
PhaseFDA-approved

Mechanism of action

Bupivacaine is a long-acting amide local anesthetic that reversibly inhibits sodium influx into nerve fibers, preventing depolarization and action potential propagation. Epinephrine is a sympathomimetic agent added to the formulation that causes vasoconstriction of local blood vessels, which slows systemic absorption of bupivacaine, extends the duration of anesthesia, and reduces the risk of systemic toxicity.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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