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Botulinum Toxin Type-A (day 0)

Ivo Pitanguy Institute · FDA-approved active Small molecule

Botulinum toxin type-A blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis.

Botulinum toxin type-A blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis. Used for Facial wrinkles and expression lines (aesthetic use), Blepharospasm, Cervical dystonia.

At a glance

Generic nameBotulinum Toxin Type-A (day 0)
SponsorIvo Pitanguy Institute
Drug classNeurotoxin; neuromuscular blocking agent
TargetSNAP-25 (synaptosome-associated protein of 25 kDa)
ModalitySmall molecule
Therapeutic areaAesthetics; Neurology; Ophthalmology
PhaseFDA-approved

Mechanism of action

The toxin is a zinc-dependent endopeptidase that cleaves SNAP-25, a protein essential for acetylcholine vesicle fusion and release. This prevents neuromuscular transmission, resulting in flaccid paralysis of injected muscles. The effect is temporary, lasting 3–4 months, after which nerve terminals regenerate and muscle function returns.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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