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Botulinum Toxin Type A (Botox)

University of Utah · FDA-approved active Small molecule

Botulinum toxin type A blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis.

Botulinum toxin type A blocks acetylcholine release at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis. Used for Cervical dystonia, Blepharospasm, Strabismus.

At a glance

Generic nameBotulinum Toxin Type A (Botox)
Also known asManual Muscle Testing (MMT), Labs (CBC, BMP, PFT'S, (FEV1 & FVC)), Iodine Starch Skin Test, BTX-A, Botulinum Neurotoxin Type A
SponsorUniversity of Utah
Drug classNeurotoxin; neuromuscular blocking agent
TargetSNAP-25 (synaptosome-associated protein of 25 kDa)
ModalitySmall molecule
Therapeutic areaNeurology; Dermatology; Aesthetics
PhaseFDA-approved

Mechanism of action

The toxin enters motor neurons and cleaves SNAP-25, a protein essential for acetylcholine vesicle fusion and release. This prevents neurotransmitter signaling to muscle fibers, resulting in localized muscle relaxation that typically lasts 3-4 months. The effect is reversible as the body gradually regenerates the cleaved protein and forms new neuromuscular connections.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results