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Botulinum neurotoxin (BTX)

The University of Texas Health Science Center, Houston · FDA-approved active Small molecule

Botulinum neurotoxin blocks the release of acetylcholine at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis.

Botulinum neurotoxin blocks the release of acetylcholine at the neuromuscular junction by cleaving SNARE proteins, causing temporary muscle paralysis. Used for Cervical dystonia, Blepharospasm, Hemifacial spasm.

At a glance

Generic nameBotulinum neurotoxin (BTX)
Also known asBotox
SponsorThe University of Texas Health Science Center, Houston
Drug classNeurotoxin; acetylcholine release inhibitor
TargetSNARE proteins (SNAP-25, VAMP, syntaxin)
ModalitySmall molecule
Therapeutic areaNeurology; Aesthetics; Pain Management
PhaseFDA-approved

Mechanism of action

BTX is a bacterial toxin that irreversibly cleaves proteins (SNAP-25, VAMP, or syntaxin) required for acetylcholine vesicle fusion and release at the presynaptic terminal. This results in temporary denervation and muscle weakness lasting 3–4 months, after which new neuromuscular junctions form and function is restored. The effect is localized to the injection site.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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