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Bortezomib, Melphalan, Prednisone

Fondazione EMN Italy Onlus · Phase 3 active Small molecule

This combination regimen kills multiple myeloma cells by inhibiting the proteasome (bortezomib), damaging DNA (melphalan), and suppressing immune responses (prednisone).

This combination regimen kills multiple myeloma cells by inhibiting the proteasome (bortezomib), damaging DNA (melphalan), and suppressing immune responses (prednisone). Used for Multiple myeloma (newly diagnosed and relapsed/refractory).

At a glance

Generic nameBortezomib, Melphalan, Prednisone
Also known asBoirtezomib, velcade
SponsorFondazione EMN Italy Onlus
Drug classProteasome inhibitor + alkylating agent + corticosteroid combination
Target20S proteasome (bortezomib); DNA (melphalan); glucocorticoid receptor (prednisone)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Bortezomib is a proteasome inhibitor that prevents degradation of pro-apoptotic proteins, triggering cancer cell death. Melphalan is an alkylating agent that cross-links DNA and causes cell death. Prednisone is a corticosteroid that provides immunosuppression and anti-inflammatory effects. Together, these three agents work synergistically to induce apoptosis in myeloma cells and overcome drug resistance.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results