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Bortezomib, cyclophosphamide, thalidomide, dexamethasone

Grupo de Estudos Multicentricos em Onco-Hematologia · Phase 3 active Small molecule

This combination regimen targets multiple pathways in multiple myeloma by inhibiting proteasome function, inducing DNA damage, suppressing tumor necrosis factor, and providing immunomodulatory effects.

This combination regimen targets multiple pathways in multiple myeloma by inhibiting proteasome function, inducing DNA damage, suppressing tumor necrosis factor, and providing immunomodulatory effects. Used for Multiple myeloma (newly diagnosed or relapsed/refractory).

At a glance

Generic nameBortezomib, cyclophosphamide, thalidomide, dexamethasone
Also known asVelcade, genuxal, decadron
SponsorGrupo de Estudos Multicentricos em Onco-Hematologia
Drug classCombination chemotherapy regimen (proteasome inhibitor + alkylating agent + immunomodulatory agent + corticosteroid)
TargetProteasome (bortezomib); DNA (cyclophosphamide); TNF-α pathway and cereblon (thalidomide); glucocorticoid receptor (dexamethasone)
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Bortezomib is a proteasome inhibitor that blocks NF-κB signaling and induces apoptosis in myeloma cells. Cyclophosphamide is an alkylating agent causing DNA damage. Thalidomide is an immunomodulatory drug (IMiD) that enhances T-cell proliferation and has anti-angiogenic properties. Dexamethasone is a corticosteroid that enhances the anti-myeloma effects of the other agents.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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