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Bevacizumab and Capecitabine

Central European Cooperative Oncology Group · Phase 3 active Biologic

Bevacizumab inhibits tumor angiogenesis by blocking VEGF, while capecitabine is a fluoropyrimidine that inhibits thymidylate synthase to disrupt DNA synthesis in cancer cells.

Bevacizumab inhibits tumor angiogenesis by blocking VEGF, while capecitabine is a fluoropyrimidine that inhibits thymidylate synthase to disrupt DNA synthesis in cancer cells. Used for Metastatic colorectal cancer, Metastatic breast cancer, Gastric cancer.

At a glance

Generic nameBevacizumab and Capecitabine
SponsorCentral European Cooperative Oncology Group
Drug classCombination therapy: monoclonal antibody (anti-VEGF) and fluoropyrimidine chemotherapy
TargetVEGF (bevacizumab); thymidylate synthase (capecitabine)
ModalityBiologic
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Bevacizumab is a monoclonal antibody that binds vascular endothelial growth factor (VEGF), preventing new blood vessel formation that tumors depend on for growth and metastasis. Capecitabine is a prodrug that is converted to 5-fluorouracil, which inhibits thymidylate synthase and disrupts nucleotide synthesis, leading to cell death. Together, they provide complementary mechanisms: anti-angiogenic therapy combined with cytotoxic chemotherapy.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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