Last reviewed · How we verify

AZILSARTAN

discontinued Small molecule

12.1 Mechanism of Action The active ingredients of Edarbyclor target two separate mechanisms involved in blood pressure regulation.

AZILSARTAN was discontinued before reaching advanced clinical trials. The drug was intended to treat high blood pressure. Its clinical significance is limited as it never progressed to a stage where it could be used to treat patients.

At a glance

Generic nameAZILSARTAN
TargetType-1 angiotensin II receptor, Type-1 angiotensin II receptor
ModalitySmall molecule
Therapeutic areaCardiovascular
Phasediscontinued

Mechanism of action

12.1 Mechanism of Action The active ingredients of Edarbyclor target two separate mechanisms involved in blood pressure regulation. Azilsartan medoxomil Angiotensin II is formed from angiotensin I in a reaction catalyzed by angiotensin-converting enzymes (ACE, kinase II). Angiotensin II is the principle pressor agent of the renin-angiotensin system, with effects that include vasoconstriction, stimulation of synthesis and release of aldosterone, cardiac stimulation, and renal reabsorption of sodi

Approved indications

Boxed warnings

Common side effects

Drug interactions

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: