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ATRA+Arsenic

Shanghai Jiao Tong University School of Medicine · FDA-approved active Small molecule

ATRA (all-trans retinoic acid) induces differentiation of leukemic cells while arsenic trioxide causes apoptosis and degradation of the PML-RARA fusion protein, together achieving complete remission in acute promyelocytic leukemia.

ATRA (all-trans retinoic acid) induces differentiation of leukemic cells while arsenic trioxide causes apoptosis and degradation of the PML-RARA fusion protein, together achieving complete remission in acute promyelocytic leukemia. Used for Acute promyelocytic leukemia (APL), newly diagnosed and relapsed/refractory.

At a glance

Generic nameATRA+Arsenic
Also known asretinoic acid + arsenic trioxide
SponsorShanghai Jiao Tong University School of Medicine
Drug classRetinoid and metalloid combination
TargetRetinoic acid receptor (RAR) and PML-RARA fusion protein
ModalitySmall molecule
Therapeutic areaOncology
PhaseFDA-approved

Mechanism of action

ATRA binds to retinoic acid receptors and promotes differentiation of abnormal promyelocytes into mature granulocytes. Arsenic trioxide induces apoptosis of leukemic cells and triggers degradation of the pathogenic PML-RARA fusion protein characteristic of APL. The synergistic combination achieves high cure rates with reduced toxicity compared to chemotherapy alone.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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