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Atovaquone Oral Suspension

University of Oxford · FDA-approved active Small molecule

Atovaquone inhibits the cytochrome bc1 complex in the mitochondrial electron transport chain of parasites, disrupting energy production and causing parasite death.

Atovaquone inhibits the cytochrome bc1 complex in the mitochondrial electron transport chain of parasites, disrupting energy production and causing parasite death. Used for Pneumocystis jirovecii pneumonia (PCP) prophylaxis and treatment, Malaria (Plasmodium falciparum) treatment, particularly in combination with proguanil.

At a glance

Generic nameAtovaquone Oral Suspension
Also known asWellvone
SponsorUniversity of Oxford
Drug classAntiparasitic agent
TargetCytochrome bc1 complex (Complex III)
ModalitySmall molecule
Therapeutic areaInfectious Disease
PhaseFDA-approved

Mechanism of action

Atovaquone is a hydroxynaphthoquinone that selectively targets the mitochondrial respiratory chain of Plasmodium species and Pneumocystis jirovecii. By inhibiting the cytochrome bc1 complex (Complex III), it prevents electron transport and collapses the mitochondrial membrane potential, leading to rapid ATP depletion and parasite death. This mechanism is particularly effective against these organisms while having minimal impact on human mitochondrial function due to structural differences in the target enzyme.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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