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ATG Fresenius

University Hospital, Basel, Switzerland · FDA-approved active Small molecule

ATG Fresenius is a polyclonal antithymocyte globulin that depletes T lymphocytes by binding to T-cell antigens and triggering complement-mediated and antibody-dependent cellular cytotoxicity.

ATG Fresenius is a polyclonal antithymocyte globulin that depletes T lymphocytes by binding to T-cell antigens and triggering complement-mediated and antibody-dependent cellular cytotoxicity. Used for Prevention of acute organ rejection in allogeneic transplantation, Treatment of aplastic anemia, Treatment of acute graft-versus-host disease.

At a glance

Generic nameATG Fresenius
Also known asSimulect IV 40 mg/day D0 and D4, and oral use Tacrolimus 0.1 mg/ kg/ day +, Myfortic 720 to 1440mg +, Corticosteroids 5mg
SponsorUniversity Hospital, Basel, Switzerland
Drug classPolyclonal antithymocyte globulin (ATG)
TargetT-cell surface antigens (CD2, CD3, CD4, CD8, CD25, HLA-DR)
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

ATG Fresenius is derived from rabbit serum immunized against human thymocytes. It contains polyclonal antibodies that recognize multiple epitopes on T cells, leading to T-cell depletion through complement activation and opsonization. This immunosuppressive effect is used to prevent graft rejection and treat aplastic anemia by reducing the T-cell mediated immune response.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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