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Aspirin tablets

PLx Pharma · FDA-approved active Small molecule

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes, reducing the production of prostaglandins and thromboxane to provide anti-inflammatory, analgesic, and antiplatelet effects.

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes, reducing the production of prostaglandins and thromboxane to provide anti-inflammatory, analgesic, and antiplatelet effects. Used for Acute myocardial infarction (secondary prevention), Ischemic stroke and transient ischemic attack (secondary prevention), Stable and unstable angina.

At a glance

Generic nameAspirin tablets
Also known asASA
SponsorPLx Pharma
Drug classNonsteroidal anti-inflammatory drug (NSAID); antiplatelet agent
TargetCyclooxygenase-1 (COX-1); Cyclooxygenase-2 (COX-2)
ModalitySmall molecule
Therapeutic areaCardiovascular; Pain management; Inflammation
PhaseFDA-approved

Mechanism of action

Aspirin acetylates serine residues on COX-1 and COX-2 enzymes, permanently blocking their catalytic activity. This reduces synthesis of prostaglandins (which mediate inflammation and pain) and thromboxane A2 (which promotes platelet aggregation). The antiplatelet effect is particularly durable because aspirin irreversibly modifies platelet COX-1, and platelets cannot synthesize new enzyme.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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