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Aspirin 81 enteric coated tablet daily

University of Florida · FDA-approved active Small molecule

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes, reducing the production of thromboxane A2 and preventing platelet aggregation.

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes, reducing the production of thromboxane A2 and preventing platelet aggregation. Used for Cardiovascular disease prevention (secondary prevention post-myocardial infarction or stroke), Acute coronary syndrome, Atrial fibrillation for stroke prevention.

At a glance

Generic nameAspirin 81 enteric coated tablet daily
Also known asAspirin
SponsorUniversity of Florida
Drug classAntiplatelet agent / Nonsteroidal anti-inflammatory drug (NSAID)
TargetCyclooxygenase-1 (COX-1)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Aspirin acetylates serine residues on COX-1 and COX-2 enzymes, permanently blocking their catalytic activity. This prevents the synthesis of thromboxane A2 in platelets, which is a key mediator of platelet activation and clot formation. At low doses (81 mg), aspirin preferentially affects platelet function, making it effective for antiplatelet therapy without significantly affecting other prostaglandin-dependent processes.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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