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Antipseudomonal beta-lactam antibiotic

Fundación Pública Andaluza para la gestión de la Investigación en Sevilla · Phase 3 active Small molecule

Inhibits bacterial cell wall synthesis by binding to penicillin-binding proteins, leading to cell wall rupture and bacterial death, with activity against Pseudomonas aeruginosa and other gram-negative organisms.

Inhibits bacterial cell wall synthesis by binding to penicillin-binding proteins, leading to cell wall rupture and bacterial death, with activity against Pseudomonas aeruginosa and other gram-negative organisms. Used for Pseudomonas aeruginosa infections (respiratory, urinary, wound, bloodstream), Nosocomial/hospital-acquired infections caused by gram-negative organisms.

At a glance

Generic nameAntipseudomonal beta-lactam antibiotic
Also known asAmpicillin, Trimethoprim/sulfamethoxazole, Cefuroxime, Amoxicillin/clavulanate, Ciprofloxacin
SponsorFundación Pública Andaluza para la gestión de la Investigación en Sevilla
Drug classAntipseudomonal beta-lactam antibiotic
TargetPenicillin-binding proteins (PBPs)
ModalitySmall molecule
Therapeutic areaInfectious Disease
PhasePhase 3

Mechanism of action

Antipseudomonal beta-lactams are beta-lactam antibiotics engineered to penetrate and remain stable against the outer membrane of Pseudomonas aeruginosa while inhibiting cross-linking of peptidoglycan in the bacterial cell wall. This class includes agents such as piperacillin, ceftazidime, and carbapenems, which are designed to overcome the intrinsic resistance mechanisms of Pseudomonas. The resulting cell wall instability causes bacterial lysis and death.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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