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anti-VEGF agent

Medical University of Vienna · FDA-approved active Small molecule

Anti-VEGF agents block vascular endothelial growth factor signaling to inhibit abnormal blood vessel formation and reduce tumor angiogenesis.

Anti-VEGF agents block vascular endothelial growth factor signaling to inhibit abnormal blood vessel formation and reduce tumor angiogenesis. Used for Metastatic colorectal cancer, Non-small cell lung cancer, Metastatic breast cancer.

At a glance

Generic nameanti-VEGF agent
Also known asCohort 1 - Quantitative, Cohort 2 - Qualitative
SponsorMedical University of Vienna
Drug classAnti-angiogenic agent (VEGF inhibitor)
TargetVEGF (Vascular Endothelial Growth Factor) and/or VEGF receptors
ModalitySmall molecule
Therapeutic areaOncology, Ophthalmology
PhaseFDA-approved

Mechanism of action

VEGF is a key driver of neovascularization in tumors and certain ocular diseases. By neutralizing VEGF or its receptors, anti-VEGF agents starve tumors of blood supply and reduce pathological vessel growth. This class includes monoclonal antibodies (e.g., bevacizumab), receptor tyrosine kinase inhibitors, and other biologics targeting the VEGF pathway.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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