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anti-pseudomonal penicillin

Vanderbilt University Medical Center · FDA-approved active Small molecule

Anti-pseudomonal penicillins inhibit bacterial cell wall synthesis by binding to penicillin-binding proteins and preventing cross-linking of peptidoglycan, with enhanced activity against Pseudomonas aeruginosa compared to standard penicillins.

Anti-pseudomonal penicillins inhibit bacterial cell wall synthesis by binding to penicillin-binding proteins and preventing cross-linking of peptidoglycan, with enhanced activity against Pseudomonas aeruginosa compared to standard penicillins. Used for Serious infections caused by Pseudomonas aeruginosa and other gram-negative bacteria, Hospital-acquired pneumonia, Febrile neutropenia in immunocompromised patients.

At a glance

Generic nameanti-pseudomonal penicillin
SponsorVanderbilt University Medical Center
Drug classBeta-lactam antibiotic (anti-pseudomonal penicillin)
TargetPenicillin-binding proteins (PBPs); bacterial cell wall peptidoglycan
ModalitySmall molecule
Therapeutic areaInfectious Disease
PhaseFDA-approved

Mechanism of action

These beta-lactam antibiotics work by disrupting bacterial cell wall integrity through inhibition of peptidoglycan cross-linking. Anti-pseudomonal penicillins (such as piperacillin and ticarcillin) have broader gram-negative coverage and greater activity against Pseudomonas aeruginosa than aminopenicillins, making them suitable for serious infections caused by this opportunistic pathogen. They are often combined with beta-lactamase inhibitors to extend coverage against beta-lactamase-producing organisms.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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