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Alpha1-Proteinase Inhibitor

Baxalta now part of Shire · FDA-approved active Biologic

Alpha1-proteinase inhibitor (AAT) replaces deficient or dysfunctional alpha-1 antitrypsin protein to protect lung tissue from enzymatic degradation.

Alpha1-proteinase inhibitor (AAT) replaces deficient or dysfunctional alpha-1 antitrypsin protein to protect lung tissue from enzymatic degradation. Used for Alpha-1 antitrypsin deficiency with emphysema or chronic obstructive pulmonary disease (COPD), Alpha-1 antitrypsin deficiency with progressive lung disease.

At a glance

Generic nameAlpha1-Proteinase Inhibitor
Also known asARALAST Fr.IV-1, ARALAST NP, GLASSIA, Zemaira®, Aralast NP
SponsorBaxalta now part of Shire
Drug classProtease inhibitor (protein replacement therapy)
TargetNeutrophil elastase (indirect target via AAT inhibition)
ModalityBiologic
Therapeutic areaPulmonology / Respiratory
PhaseFDA-approved

Mechanism of action

Alpha-1 antitrypsin (AAT) is a serine protease inhibitor that normally protects lung tissue from neutrophil elastase and other proteases released during inflammation. In alpha-1 antitrypsin deficiency, insufficient AAT levels allow unopposed protease activity, leading to progressive lung destruction. Exogenous AAT replacement therapy augments circulating and lung AAT levels to slow or prevent emphysema progression.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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