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Alendronate (Fosamax)

Medical University of South Carolina · FDA-approved active Small molecule

Alendronate inhibits osteoclast-mediated bone resorption by binding to hydroxyapatite on bone surfaces and blocking the enzyme farnesyl pyrophosphate synthase in the mevalonate pathway.

Alendronate inhibits osteoclast-mediated bone resorption by binding to hydroxyapatite on bone surfaces and blocking the enzyme farnesyl pyrophosphate synthase in the mevalonate pathway. Used for Osteoporosis in postmenopausal women, Osteoporosis in men, Glucocorticoid-induced osteoporosis.

At a glance

Generic nameAlendronate (Fosamax)
Also known asFosamax
SponsorMedical University of South Carolina
Drug classBisphosphonate
TargetFarnesyl pyrophosphate synthase (FPPS); hydroxyapatite binding
ModalitySmall molecule
Therapeutic areaBone metabolism / Rheumatology
PhaseFDA-approved

Mechanism of action

As a bisphosphonate, alendronate is selectively taken up by osteoclasts during bone resorption. Once internalized, it inhibits farnesyl pyrophosphate synthase, disrupting the prenylation of small GTPases required for osteoclast function and survival. This leads to decreased bone turnover and increased bone mineral density, making it effective for conditions characterized by excessive bone loss.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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