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aflibercept + FOLFIRI

CR-CSSS Champlain-Charles-Le Moyne · FDA-approved active Small molecule

Aflibercept blocks VEGF signaling to inhibit tumor angiogenesis, while FOLFIRI chemotherapy directly kills cancer cells through fluorouracil and irinotecan.

Aflibercept blocks VEGF signaling to inhibit tumor angiogenesis, while FOLFIRI chemotherapy directly kills cancer cells through fluorouracil and irinotecan. Used for Metastatic colorectal cancer (in combination with FOLFIRI), Second-line treatment of metastatic colorectal cancer.

At a glance

Generic nameaflibercept + FOLFIRI
Also known asaflibercept, FOLFIRI
SponsorCR-CSSS Champlain-Charles-Le Moyne
Drug classVEGF inhibitor (fusion protein) + chemotherapy combination
TargetVEGF-A, VEGF-B, PlGF (aflibercept component); topoisomerase I and thymidylate synthase (FOLFIRI component)
ModalitySmall molecule
Therapeutic areaOncology
PhaseFDA-approved

Mechanism of action

Aflibercept is a VEGF trap that sequesters vascular endothelial growth factor, preventing new blood vessel formation that tumors depend on for growth. FOLFIRI is a combination chemotherapy regimen (5-fluorouracil, leucovorin, and irinotecan) that damages DNA and inhibits topoisomerase I. Together, they provide dual anti-angiogenic and cytotoxic activity against colorectal cancer.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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