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Administration of Vancomycin

University of Colorado, Denver · FDA-approved active Small molecule

Vancomycin inhibits bacterial cell wall synthesis by binding to D-Ala-D-Ala peptide precursors and preventing peptidoglycan cross-linking.

Vancomycin inhibits bacterial cell wall synthesis by binding to D-Ala-D-Ala peptide precursors and preventing peptidoglycan cross-linking. Used for Serious infections caused by susceptible strains of methicillin-resistant Staphylococcus aureus (MRSA), Clostridium difficile-associated diarrhea and colitis, Endocarditis caused by susceptible streptococci or staphylococci.

At a glance

Generic nameAdministration of Vancomycin
Also known asVancomycin Hydrochloride
SponsorUniversity of Colorado, Denver
Drug classGlycopeptide antibiotic
TargetD-Ala-D-Ala peptidoglycan precursor
ModalitySmall molecule
Therapeutic areaInfectious Disease
PhaseFDA-approved

Mechanism of action

Vancomycin is a glycopeptide antibiotic that binds to the D-alanyl-D-alanine terminus of peptidoglycan precursors in bacterial cell walls, blocking the cross-linking of peptidoglycan strands. This disrupts cell wall integrity and leads to bacterial cell lysis and death. It is bactericidal and particularly effective against gram-positive organisms.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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