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Adalimumab ID

Centre for Human Drug Research, Netherlands · Phase 1 active Biologic Under review

Adalimumab ID is a TNF-alpha inhibitor Biologic drug developed by Centre for Human Drug Research, Netherlands. It is currently in Phase 1 development for Rheumatoid arthritis, Psoriatic arthritis, Ankylosing spondylitis.

Tumour necrosis factor-alpha inhibitor

Adalimumab ID is a microneedle delivery method for the treatment of pain and injection site conditions, compared to subcutaneous injection. Adalimumab ID is a TNF-alpha inhibitor, classified as an antibody inhibitor, targeting tumor necrosis factor.

Likelihood of approval
10.6% vs 9.6% industry baseline
If approved by FDA: likely 2033–2036
Steps remaining: Phase 2 → Phase 3 → NDA/BLA submission
Confidence: Medium
Why this estimate
  • Baseline phase 1 → approval rate +9.6pp
    Industry-wide phase 1 drugs reach approval ~9.6% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Immunology slight uplift +1.0pp
    Mature endpoint landscape (ACR, DAS28, PASI) makes immunology approvals slightly more predictable.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2033–2036
EMA EU 2034–2037 +0.7 yr
MHRA GB 2034–2037 +0.7 yr
Health Canada CA 2034–2038 +0.9 yr
TGA AU 2034–2038 +1.2 yr
PMDA JP 2034–2038 +1.5 yr
NMPA CN 2035–2039 +2.3 yr
MFDS KR 2034–2038 +1.4 yr
CDSCO IN 2034–2039 +1.8 yr
ANVISA BR 2035–2039 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic nameAdalimumab ID
SponsorCentre for Human Drug Research, Netherlands
Drug classTNF-alpha inhibitor
TargetTumour necrosis factor-alpha
ModalityBiologic
Therapeutic areaImmunology
PhasePhase 1

Mechanism of action

Adalimumab is a fully human monoclonal antibody that binds to tumour necrosis factor-alpha (TNF-alpha), preventing its interaction with cell surface TNF receptors and subsequent inflammatory response.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about Adalimumab ID

What is Adalimumab ID?

Adalimumab ID is a TNF-alpha inhibitor drug developed by Centre for Human Drug Research, Netherlands, indicated for Rheumatoid arthritis, Psoriatic arthritis, Ankylosing spondylitis.

How does Adalimumab ID work?

Tumour necrosis factor-alpha inhibitor

What is Adalimumab ID used for?

Adalimumab ID is indicated for Rheumatoid arthritis, Psoriatic arthritis, Ankylosing spondylitis, Crohn's disease, Ulcerative colitis.

Who makes Adalimumab ID?

Adalimumab ID is developed by Centre for Human Drug Research, Netherlands (see full Centre for Human Drug Research, Netherlands pipeline at /company/centre-for-human-drug-research-netherlands).

What drug class is Adalimumab ID in?

Adalimumab ID belongs to the TNF-alpha inhibitor class. See all TNF-alpha inhibitor drugs at /class/tnf-alpha-inhibitor.

What development phase is Adalimumab ID in?

Adalimumab ID is in Phase 1.

What are the side effects of Adalimumab ID?

Common side effects of Adalimumab ID include Injection site reaction, Headache, Fatigue, Nausea, Diarrhea.

What does Adalimumab ID target?

Adalimumab ID targets Tumour necrosis factor-alpha and is a TNF-alpha inhibitor.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing