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ACE Inhibitors and Diuretics

The University of Texas Health Science Center, Houston · FDA-approved active Small molecule

ACE inhibitors block angiotensin-converting enzyme to reduce angiotensin II production and lower blood pressure, while diuretics increase sodium and water excretion to reduce fluid volume and blood pressure.

ACE inhibitors block angiotensin-converting enzyme to reduce angiotensin II production and lower blood pressure, while diuretics increase sodium and water excretion to reduce blood volume and fluid overload. Used for Hypertension, Heart failure with reduced ejection fraction, Post-myocardial infarction.

At a glance

Generic nameACE Inhibitors and Diuretics
SponsorThe University of Texas Health Science Center, Houston
Drug classACE inhibitor + diuretic combination
TargetAngiotensin-converting enzyme (ACE); various diuretic targets depending on class (e.g., loop of Henle, distal convoluted tubule)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

ACE inhibitors prevent the conversion of angiotensin I to angiotensin II, a potent vasoconstrictor, thereby reducing peripheral vascular resistance and blood pressure. Diuretics work through different mechanisms (thiazide, loop, or potassium-sparing) to promote renal sodium and water excretion, decreasing circulating blood volume. The combination provides complementary antihypertensive effects and is commonly used in fixed-dose formulations for improved patient adherence.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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