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ACE inhibitor

University of Cambridge · FDA-approved active Small molecule

ACE inhibitors block the angiotensin-converting enzyme, preventing the conversion of angiotensin I to angiotensin II and thereby reducing vasoconstriction and aldosterone release.

ACE inhibitors block the angiotensin-converting enzyme, preventing the conversion of angiotensin I to angiotensin II and thereby reducing vasoconstriction and aldosterone release. Used for Hypertension, Heart failure, Post-myocardial infarction.

At a glance

Generic nameACE inhibitor
Also known asQuinapril, Aspirin , statins, Captopril or equivalent, conservative treatment, Angiotensin-converting enzyme blocker
SponsorUniversity of Cambridge
Drug classACE inhibitor
TargetAngiotensin-converting enzyme (ACE)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

By inhibiting ACE, these drugs lower levels of angiotensin II, a potent vasoconstrictor and hormone that increases blood pressure and promotes sodium retention. This leads to vasodilation, reduced blood pressure, decreased cardiac workload, and improved renal perfusion. The mechanism is particularly beneficial in hypertension and heart failure by reducing both preload and afterload on the heart.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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