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Abemaciclib + Fulvestrant

Prof. Wolfgang Janni · FDA-approved active Small molecule

Abemaciclib inhibits CDK4/6 to block cell cycle progression, while fulvestrant is an estrogen receptor antagonist that degrades the ER protein, together blocking hormone-driven breast cancer growth.

Abemaciclib inhibits CDK4/6 to block cell cycle progression, while fulvestrant is an estrogen receptor antagonist that degrades the ER protein, together blocking hormone-driven breast cancer growth. Used for Hormone receptor-positive, HER2-negative advanced or metastatic breast cancer in postmenopausal women, Hormone receptor-positive, HER2-negative advanced or metastatic breast cancer in premenopausal women (with endocrine therapy).

At a glance

Generic nameAbemaciclib + Fulvestrant
Also known asVerzenios
SponsorProf. Wolfgang Janni
Drug classCDK4/6 inhibitor + estrogen receptor antagonist
TargetCDK4/6 and estrogen receptor (ER)
ModalitySmall molecule
Therapeutic areaOncology
PhaseFDA-approved

Mechanism of action

Abemaciclib is a selective CDK4/6 inhibitor that prevents phosphorylation of the retinoblastoma protein, halting G1/S cell cycle transition in hormone receptor-positive breast cancer cells. Fulvestrant is a selective estrogen receptor degrader (SERD) that binds to and promotes degradation of the estrogen receptor, eliminating ER-mediated signaling. The combination provides dual blockade of cell cycle and estrogen signaling pathways.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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