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AAT

Rabin Medical Center · FDA-approved active Small molecule

AAT (Alpha-1 Antitrypsin) is a protease inhibitor that replaces deficient endogenous alpha-1 antitrypsin to protect lung tissue from enzymatic degradation.

AAT is an alpha-1 antitrypsin replacement therapy that replenishes deficient serum levels of the protease inhibitor to prevent progressive lung and liver damage. Used for Alpha-1 antitrypsin deficiency with emphysema, Alpha-1 antitrypsin deficiency with progressive lung disease.

At a glance

Generic nameAAT
Also known asAlpha-1 proteinase inhibitor, Respreeza
SponsorRabin Medical Center
Drug classProtease inhibitor replacement therapy
TargetAlpha-1 antitrypsin (serum protein replacement)
ModalitySmall molecule
Therapeutic areaPulmonology / Hepatology
PhaseFDA-approved

Mechanism of action

Alpha-1 antitrypsin deficiency leads to unopposed neutrophil elastase activity in the lungs, causing progressive tissue destruction. AAT therapy restores the protease-antiprotease balance by supplying exogenous alpha-1 antitrypsin, which inhibits neutrophil elastase and other serine proteases, thereby slowing lung parenchymal damage and decline in pulmonary function.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results